In-Degree and PageRank of Web pages: Why do they follow similar power laws?

The PageRank is a popularity measure designed by Google to rank Web pages. Experiments confirm that the PageRank obeys a `power law' with the same exponent as the In-Degree. This paper presents a novel mathematical model that explains this phenomenon. The relation between the PageRank and In-Degree is modelled through a stochastic equation, which is inspired by the original definition of the PageRank, and is analogous to the well-known distributional identity for the busy period in the M/G/1 queue. Further, we employ the theory of regular variation and Tauberian theorems to analytically prove that the tail behavior of the PageRank and the In-Degree differ only by a multiplicative factor, for which we derive a closed-form expression. Our analytical results are in good agreement with experimental data.Comment: 20 pages, 3 figures; typos added; reference adde

Spectral properties of the tandem Jackson network, seen as a quasi-birth-and-death process

Quasi-birth-and-death (QBD) processes with infinite ``phase spaces'' can exhibit unusual and interesting behavior. One of the simplest examples of such a process is the two-node tandem Jackson network, with the ``phase'' giving the state of the first queue and the ``level'' giving the state of the second queue. In this paper, we undertake an extensive analysis of the properties of this QBD. In particular, we investigate the spectral properties of Neuts's R-matrix and show that the decay rate of the stationary distribution of the ``level'' process is not always equal to the convergence norm of R. In fact, we show that we can obtain any decay rate from a certain range by controlling only the transition structure at level zero, which is independent of R. We also consider the sequence of tandem queues that is constructed by restricting the waiting room of the first queue to some finite capacity, and then allowing this capacity to increase to infinity. We show that the decay rates for the finite truncations converge to a value, which is not necessarily the decay rate in the infinite waiting room case. Finally, we show that the probability that the process hits level n before level 0 given that it starts in level 1 decays at a rate which is not necessarily the same as the decay rate for the stationary distribution.Comment: Published at http://dx.doi.org/10.1214/105051604000000477 in the Annals of Applied Probability (http://www.imstat.org/aap/) by the Institute of Mathematical Statistics (http://www.imstat.org

A Stochastic Fluid Model Approach to the Stationary Distribution of the Maximum Priority Process

In traditional priority queues, we assume that every customer upon arrival has a fixed, class-dependent priority, and that a customer may not commence service if a customer with a higher priority is present in the queue. However, in situations where a performance target in terms of the tails of the class-dependent waiting time distributions has to be met, such models of priority queueing may not be satisfactory. In fact, there could be situations where high priority classes easily meet their performance target for the maximum waiting time, while lower classes do not. Here, we are interested in the stationary distribution at the times of commencement of service of this maximum priority process. Until now, there has been no explicit expression for this distribution. We construct a mapping of the maximum priority process to a tandem fluid queue, which enables us to find expressions for this stationary distribution. We derive the results for the stationary distribution of the maximum priority process at the times of the commencement of service.Comment: The Eleventh International Conference on Matrix-Analytic Methods in Stochastic Models (MAM11), 2022, Seoul, Republic of Kore

Novel multiple sclerosis susceptibility loci implicated in epigenetic regulation

We conducted a genome-wide association study (GWAS) on multiple sclerosis (MS) susceptibility in German cohorts with 4888 cases and 10,395 controls. In addition to associations within the major histocompatibility complex (MHC) region, 15 non-MHC loci reached genome-wide significance. Four of these loci are novel MS susceptibility loci. They map to the genes L3MBTL3, MAZ, ERG, and SHMT1. The lead variant at SHMT1 was replicated in an independent Sardinian cohort. Products of the genes L3MBTL3, MAZ, and ERG play important roles in immune cell regulation. SHMT1 encodes a serine hydroxymethyltransferase catalyzing the transfer of a carbon unit to the folate cycle. This reaction is required for regulation of methylation homeostasis, which is important for establishment and maintenance of epigenetic signatures. Our GWAS approach in a defined population with limited genetic substructure detected associations not found in larger, more heterogeneous cohorts, thus providing new clues regarding MS pathogenesis